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These are, in effect, competing engineering goals, because the hydrophobic modifications that extend half-life can also promote aggregation

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10.1002/ddr.21737 45 KawaguchiM.KataokaH

At the same time, in glutathione-treated old rats, we also observed restoration of endothelium-dependent vasorelaxation responses to acetylcholine, which were almost completely abolished by the NO-synthase inhibitor L-NAME

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